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The TRPC5 receptor as pharmacological target for pain and metabolic disease

Tong, R

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Broader solid tumor studies corroborate that pharmacological inhibition of CAF-mediated Gln synthesis disrupts tumor metabolic fitness, suggesting combinatorial targeting of tumoral and stromal glutamine metabolism, particularly through dual GLS1/ECM pathway inhibition, may offer promising therapeutic avenues for advanced prostate cancer (196)

Studies have shown that people who live well into their 80s and beyond have higher levels of glutathione