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These endocrine abnormalities if not treated, culminate to metabolic syndrome development, in which the key pathophysiological components include abdominal fat accumulation, reduced systemic insulin sensitivity, low-grade inflammation, high blood pressure, high blood sugar levels, high triglyceride levels, and low levels of high-density lipoprotein (HDL) cholesterol [2]

In tumor tissue, intermittent blood supply leads to an alternating situation of lack-of-oxygen and reoxygenation, which causes oxidative stress

Whether these mutations have a causative or associative role in the ASD phenotype remains to be determined

Arenas J, Ricoy JR, Encinas AR, Pola P, DIddio S, Zeviani M, Didonato S, Corsi M

Garzya G, et al

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