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Mito-TEMPO (Sigma-Aldrich, Milan, Italy) was solubilized in DMSO and added to the cells simultaneously with the medium change 24 h before the viability assay

Results DMF activates the TSS pathway in SCD erythroblasts To investigate the mechanism of DMF on ferroptosis regulation, we conducted untargeted LC-MS using erythroblasts generated from CD34+ stem cells isolated from the peripheral blood of SCD patient

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Available treatments such as levodopa, dopamine agonists and monoamine oxidase B inhibitors offer only symptomatic relief however, they do not treat the underlying neurodegeneration, emphasising the need for neuroprotective management

To obtain a thorough knowledge of the capabilities of targeting moieties in treating GBM, additional research is imperative