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Fluoxetine and fluvoxamine are potent and hit a broad array of P450 enzymes, while paroxetines effects are fairly specific for CYP2D6

Molecular alterations in receptor functionality, particularly IR signaling and GLUT4 translocation, generate metabolic disruptions through complex inter-organ communication

The activation of IGF-1R triggers multiple signal cascades in a cell context dependent manner to regulate multiple cellular functions from proliferation and survival to differentiation into specific cell lineages[39]

CD4 + T cells are divided into different sub-populations, such as Th1 and Th2, based on the type of cytokines produced

GS is expressed in utero in early human fetuses[9] and is a prerequisite for early development in mice and drosophila[10, 11]

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