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In separate studies, Song, Lapin, and colleagues demonstrated that condensation of TIR domain-containing proteins elucidates LLPS-mediated promotion of plant RCD: upon pathogen recognition, upstream TIR proteins form condensates that scaffold and amplify enzymatic activity, with the signal being subsequently relayed to the EDS1-associated death module, thereby driving the execution of hypersensitive response (HR)-type RCD [113, 114]