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Recombinant active SRMS was a gift from D

Clinical data, dosing protocols, and Canadian access

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RNA-binding elements Although charge-reversal mutations within RBE2 and RBE3 did not individually impact rescue of miR-7 degradation in ZSWIM8-deficient cells, combined mutation of all three RBEs exacerbated the defect caused by mutating RBE1, leading to severe loss of both TDMD in cells and polyubiquitylation in vitro (Fig