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[Google Scholar] Zhang, Q

RSC Advances, 13(48): 3395733993 Grimsey E M, Fais C, Marshall R L, Ricci V, Ciusa M L, Stone J W, Ivens A, Malloci G, Ruggerone P, Vargiu A V, et al

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On the other hand, rifampicin exhibits its toxicity by activating pregnane X receptor (PXR), leading to an increased generation of reactive intermediates with hepatotoxic and nephrotoxic potential [5,6,7]

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Accordingly, it was observed that, while stimulation with TNF and IL-1 induced the transcription of NF-B target genes, as well as its binding to their promoters, in p53 KO cells, the cytokine-driven transcriptional activation of NF-B was abolished in p53 KO Cry DKO cells