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Research Background This study focuses on the biological functions and molecular mechanisms of METTL14 (Methyltransferase 14, N6-Adenosine-Methyltransferase Subunit) in colorectal cancer (CRC)

A possible justification for the modification of BSA/AuNCs was to enhance aurophilic interactions of Au(I)-thiolate complexes on the surface of Au(0) core and rigidify the ligand shell

This targeted delivery mechanism could revolutionize how neuroprotective compounds reach the exact cells that need them most

To evaluate the role the CBM loop plays in UNC10201652 inhibition, we deleted residues N737 to Y499 that form the CBM loop in Bd GUS

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