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It has been noted (11, 12) that NADPH oxidase-mediated oxidative stress and P53 (p53 is a tumor suppressor protein that regulates the expression of a variety of genes that are involved in apoptosis, growth arrest, inhibition of cell cycle progression, differentiation and accelerated DNA repair or senescence in response to genotoxicity or cell stress.) and Bax/Bcl2-mediated apoptosis signaling pathways are involved in the pathogenesis of cataracts in diabetic rats, and Puerarin may exert a therapeutic effect on diabetic cataract rats by improving the state of insulin resistance and downregulating the level of P53 protein (12)

The expressions of ATX, LPAR2, and LPAR6 were detected by immunohistochemistry (IHC)

Specifically, direct repair methods include base excision repair and mismatch repair

Melatonin reduces early changes in intramitochondrial cardiolipin during apoptosis in U937 cell line

However, Ueno et al

B., Samanta, D