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Kanzawa T, Zhang L, Xiao L, Germano IM, Kondo Y, Kondo S

The expression of HvGR1 was mainly localized to shoot tissues and only slight expression was observed in roots

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Consequently, deficiencies in any of these processes caused by the loss of ATM could be used as therapeutic targets.1110 Consistent with this notion, ATM-null CLL cells are defective in homologous recombination repair (HRR), a deficiency that can be exploited to induce tumor-specific killing via enhanced requirement for HRR upon PARP-inhibition.11 ATM is also implicated in redox homeostasis.12 Deregulation of redox homeostasis results in oxidative stress and occurs either due to increased reactive oxygen species (ROS) production or reduced antioxidant capacity

Environ Pollut 203:4049